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SEARCH RESULTS FOR: Open-Fractures

Offene Frakturen: Mechanismen, klinische Befunde und Komplikationen

Offene Frakturen: Mechanismen, klinische Befunde und Komplikationen

Open Fractures

Open Fractures: Mechanisms, clinical features and complications
  Direct, high-energy force (e.g. vehicle collisions, gunshot) or low-energy force on diseased bone
Force applied to bone exceeds strength of bone resulting in periosteal stripping and subsequent soft tissue and neurovascular destruction
Open Fractures
 Also known as “compound fractures” and classified with the Gustilo-Anderson classification system (Types I, II, III), these are fractures in which the skin is penetrated and bone is exposed to the external environment. Comminuted fractures have ≥ 2 breaks in the bone.
  Inside-out (bone) or outside-in (external) penetration of skin to create a wound
Skin tearing creates vacuum-like effect pulling debris into wound
       Minimal comminution
Bone penetrates skin to create a wound < 1 cm in diameter (Type I)
Smaller wound creates minimal opportunities for pathogen entry and contamination
Moderate comminution
Bone penetrates skin to create a wound 1-10 cm in diameter (Type II)
Moderate wound creates some opportunity for pathogen entry and contamination
Extensive comminution
Bone penetrates skin to create a wound > 10 cm in diameter (Type III)
Large wound creates ample opportunity for pathogen entry and extensive contamination
Type IIIA (adequate soft tissue for bone coverage)
Type IIIB
(soft tissue damage with periosteal stripping)
Type IIIC (vascular injuries, potential amputation)
Displacement/shortening/ angulation/rotation of fracture fragment
Improper bone healing
Bone deformity
Authors: Meaghan MacKenzie Holly Zahary Loreman Nojan Mannani Reviewers: Annalise Abbott Usama Malik Michelle J. Chen Dr. Prism Schneider* Dr. Jared Topham* * MD at time of publication
                  Pain & lack of mechanical load bearing axis
Inability to weight bear
Decreased mobility promotes stasis of venous blood flow & intravascular vessel wall damage
Deep vein thrombosis
Potential progression to pulmonary embolism
Bleeding or inflammation within fascia
Muscle atrophy
Compartment syndrome (↑ pressure in muscle) **
Open wound exposes bone
Infiltration of debris & contaminants
Infection of soft tissues or bone (osteomyelitis)
Initial injury damages blood vessels
Initial injury damages nerves
↓ Sensation distal to injury
↓ Limb function & proprioception
↓ Pulses distal to injury
Amputation
       ↓ Blood flow to bone
Avascular necrosis (bone tissue death)
Limb ischemia
↓ Blood flow & oxygen delivery to tissues
Compartment syndrome**
                       Delayed union (bone healing) on serial radiographs
Non-union (bone fails to heal) on serial radiographs
 **See corresponding Calgary Guide slide on Acute Compartment Syndrome
 Legend:
 Pathophysiology
Mechanism
Sign/Symptom/Lab Finding
 Complications
 Published Nov 16, 2017; updated Nov 21, 2024 on www.thecalgaryguide.com